Best adjuvant (assist) for chemotherapy | 1+1>487% |
Effectively improve chemotherapy effect, treatment, immunity |
Reduce side effects and recurrence | 
Overview / Relation / Abstract / Role / Principle / Action / Mechanism / Function / Work | 
Abstract / Summary / Overview of Apoptosis
Why do cells undergo apoptosis?
The relationship between cancer cells and apoptosis
Where are the weaknesses and symptoms of cancer cells?
Are cancer cells aggressive?
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)
Solamargine's major function mechanism:
Solamargine vs cancer
Best Chemotherapy Adjuvant  (1+1>478%) Effectively improve chemotherapy effect and cure
Best adjuvant (assist) for chemotherapy | 1+1>487%
Effectively improve chemotherapy effect, treatment, immunity 
Reduce side effects and recurrence 
Overview / Relation / Abstract / Role / Principle  / Action / Mechanism / Function / Work
Abstract / Summary / Overview of Apoptosis

Overview of apoptosis
•Programmed cell death
•Apoptosis is a form of programmed cell death, or “cellular suicide.”
•Apoptosis is different from necrosis, in which cells die due to injury.
•Apoptosis removes cells during development, eliminates potentially cancerous and virus-infected cells, and maintains balance in the body.
Why do cells undergo apoptosis?
- Basically, apoptosis is a general and convenient way to remove cells that should no longer be part of the organism.
 - Some cells are abnormal and could hurt the rest of the organism if they survive, such as cells with viral infections or DNA damage.
 - Apoptosis is part of development
 - In many organisms, programmed cell death is a normal part of development.
 
The relationship between cancer cells and apoptosis
Apoptosis can eliminate infected or cancerous cells.
When a cell’s DNA is damaged, it will typically detect the damage and try to repair it. 
If the damage is beyond repair, the cell will normally send itself into apoptosis, ensuring that it will not pass on its damaged DNA. 
When cells have DNA damage but fail to undergo apoptosis, they may be on the road to cancer.
However, “successful” cancer cells successfully evade the process of apoptosis.
This allows them to divide out of control and accumulate mutations (changes in their DNA).
Apoptosis is key to immune function
Apoptosis also plays an essential role in the development and maintenance of a healthy immune system. 
Where are the weaknesses and symptoms of cancer cells?
The symptoms of cancer cells are in the nucleus.
The nucleus controls the outer cytoplasm, cell composition, cell viability, etc.
DNA mutations also mutate in the nucleus.
Therefore, to treat cancer cells, we must first enter the nucleus.
Let the “regulatory cell gene” mechanism enter the nucleus to regulate
Are cancer cells aggressive?
After the action of Solamargine, the aggressiveness of cancer cells is alleviated.
So after using Solamargine, many patients feel that I am half better.
Although the tumor does not disappear quickly, patients feel that the degree of aggressiveness is reduced.
Extraordinary Solamargine (Role, Principle, Action, Mechanism, Function, Work)

Solamargine's major function mechanism:
When Solamargine enter, 
Solamargine activates receptors that are turned off by cancer cells, allowing cancer cells to modulate again. 
Solamargine modulates the anti-modulates genes of cancer cells, making cancer cells less resistant. 
Reduced drug resistance 
When cancer cells are less resistant to drugs, chemotherapy becomes more effective. 
Solamargine modulates the mutated genes in cancer cells and then initiates cancer cell apoptosis to achieve anti-cancer effects.
Solamargine combined with which chemotherapy drugs are more effective in treating cancer cells?

Solamargine vs cancer

Solamargine vs cancer
The picture shows the death of cancer cells.
The black and black parts are cancer cell nuclei.
Even if the nucleus ruptures, the cancer cells will die.
The figure shows that cancer cells can cause death.

The figure shows that cancer cells can cause death.
The figure shows that the death of lung cancer cells is relatively slow, and it will not be obvious until eight hours later.
The figure shows that the death of liver cancer cells is very obvious, even more obvious in eight hours.
The graph shows that breast cancer cells die faster. It was obvious from the beginning that breast cancer is easy to treat, and patients with breast cancer need not worry.
Best Chemotherapy Adjuvant (1+1>487%) Effectively improve chemotherapy effect and treatment
ANTI-CANCER 
Patent protection in 32 nations. 
A comparison study showing Solamargine vs. other therapeutic drugs with respect to lung cancer cells.

A comparison study showing Solamargine vs. other chemotherapeutic drugs with respect to breast cancer cells

SR-T100 combination therapy with effective result against breast cancer cells.

Combination Therapy   |   Research results for lung cancer cells. 
A. Chemotherapy    (100μM), 16% of cancer cell apoptosis. 
B. Alone SM (4.8μM), 28% of cancer cell apoptosis. 
C. SM (4.80μM) + Chemotherapy (40μM), 66% of cancer cells apoptosis.  
D. SM (4.80μM) + Chemotherapy (100μM), 78% of cancer cell apoptosis.  
SM has a clearing effect better than Chemotherapy. 
The combined treatment of Solamargine and Chemotherapy significantly increased the apoptosis of lung cancer cells.  
SM (4.8μM) + Chemotherapy (40μM), increased from 16% to 66% (up to 4.125 times).  
SM (4.8μM) + Chemotherapy (100μM), increased from 16% to 78% (up to 4.875 times)  
Reorganized from: BBRC. Action of Solamargine on TNFs and drug-resistant human lung cancer cells 2004

Solamargine Q&A (English)
survival rate | cancer | Contents 
survival rate | cancer | Etymology and definitions 
survival rate | cancer | Signs and symptoms 
survival rate | cancer | Local symptoms 
survival rate | cancer | Systemic symptoms 
survival rate | cancer | Metastasis 
survival rate | cancer | Causes 
survival rate | cancer | Chemicals 
survival rate | cancer | Diet and exercise 
survival rate | cancer | Infection 
survival rate | cancer | Radiation 
survival rate | cancer | Heredity 
survival rate | cancer | Physical agents 
survival rate | cancer | Hormones 
survival rate | cancer | Autoimmune diseases 
survival rate | cancer | Pathophysiology 
survival rate | cancer | Genetics 
survival rate | cancer | Epigenetics 
survival rate | cancer | Metastasis 
survival rate | cancer | Metabolism 
survival rate | cancer | Diagnosis 
survival rate | cancer | Classification 
survival rate | cancer | Prevention 
survival rate | cancer | Dietary 
survival rate | cancer | Medication 
survival rate | cancer | Vaccination 
survival rate | cancer | Screening 
survival rate | cancer | Recommendations 
survival rate | cancer | Genetic testing 
survival rate | cancer | Management 
survival rate | cancer | Cancer 
survival rate | cancer | Radiation 
survival rate | cancer | Surgery 
survival rate | cancer | Palliative care 
survival rate | cancer | Immunotherapy 
survival rate | cancer | Laser therapy 
survival rate | cancer | Alternative medicine 
survival rate | cancer | Prognosis 
survival rate | cancer | Epidemiology 
survival rate | cancer | History 
survival rate | cancer | Society and culture 
survival rate | cancer | Economic effect 
survival rate | cancer | Workplace 
survival rate | cancer | Research 
survival rate | cancer | Pregnancy 
survival rate | cancer | Other animals 
survival rate | cancer | Notes 
survival rate | cancer | Further reading 
survival rate | cancer | External links 
Ronn Zell Tumor | Iwwerliewensquote | Contents 
Ronn Zell Tumor | Iwwerliewensquote | Etymology and definitions 
Ronn Zell Tumor | Iwwerliewensquote | Signs and symptoms 
Ronn Zell Tumor | Iwwerliewensquote | Local symptoms 
Ronn Zell Tumor | Iwwerliewensquote | Systemic symptoms 
Ronn Zell Tumor | Iwwerliewensquote | Metastasis 
Ronn Zell Tumor | Iwwerliewensquote | Causes 
Ronn Zell Tumor | Iwwerliewensquote | Chemicals 
Ronn Zell Tumor | Iwwerliewensquote | Diet and exercise 
Ronn Zell Tumor | Iwwerliewensquote | Infection 
Ronn Zell Tumor | Iwwerliewensquote | Radiation 
Ronn Zell Tumor | Iwwerliewensquote | Heredity 
Ronn Zell Tumor | Iwwerliewensquote | Physical agents 
Ronn Zell Tumor | Iwwerliewensquote | Hormones 
Ronn Zell Tumor | Iwwerliewensquote | Autoimmune diseases 
Ronn Zell Tumor | Iwwerliewensquote | Pathophysiology 
Ronn Zell Tumor | Iwwerliewensquote | Genetics 
Ronn Zell Tumor | Iwwerliewensquote | Epigenetics 
Ronn Zell Tumor | Iwwerliewensquote | Metastasis 
Ronn Zell Tumor | Iwwerliewensquote | Metabolism 
Ronn Zell Tumor | Iwwerliewensquote | Diagnosis 
Ronn Zell Tumor | Iwwerliewensquote | Classification 
Ronn Zell Tumor | Iwwerliewensquote | Prevention 
Ronn Zell Tumor | Iwwerliewensquote | Dietary 
Ronn Zell Tumor | Iwwerliewensquote | Medication 
Ronn Zell Tumor | Iwwerliewensquote | Vaccination 
Ronn Zell Tumor | Iwwerliewensquote | Screening 
Ronn Zell Tumor | Iwwerliewensquote | Recommendations 
Ronn Zell Tumor | Iwwerliewensquote | Genetic testing 
Ronn Zell Tumor | Iwwerliewensquote | Management 
Ronn Zell Tumor | Iwwerliewensquote | Chemotherapy 
Ronn Zell Tumor | Iwwerliewensquote | Radiation 
Ronn Zell Tumor | Iwwerliewensquote | Surgery 
Ronn Zell Tumor | Iwwerliewensquote | Palliative care 
Ronn Zell Tumor | Iwwerliewensquote | Immunotherapy 
Ronn Zell Tumor | Iwwerliewensquote | Laser therapy 
Ronn Zell Tumor | Iwwerliewensquote | Alternative medicine 
Ronn Zell Tumor | Iwwerliewensquote | Prognosis 
Ronn Zell Tumor | Iwwerliewensquote | Epidemiology 
Ronn Zell Tumor | Iwwerliewensquote | History 
Ronn Zell Tumor | Iwwerliewensquote | Society and culture 
Ronn Zell Tumor | Iwwerliewensquote | Economic effect 
Ronn Zell Tumor | Iwwerliewensquote | Workplace 
Ronn Zell Tumor | Iwwerliewensquote | Research 
Ronn Zell Tumor | Iwwerliewensquote | Pregnancy 
Ronn Zell Tumor | Iwwerliewensquote | Other animals 
Ronn Zell Tumor | Iwwerliewensquote | Notes 
Ronn Zell Tumor | Iwwerliewensquote | Further reading 
Ronn Zell Tumor | Iwwerliewensquote | External links